Arthrogryposis Multiplex

Arthrogryposis Multiplex (AM) Information

Notice of November 20, 2008: On Saturday, November 15, 2008, the Board of Directors of the Association adopted a new policy and related set of rules governing the handling of genetic defects, including Arthrogryposis Multiplex (AM), within the breed. Click here for a complete posting of new policies and related rules.

The following summary was developed to respond to questions commonly asked by American Angus Association members. Click here.

A video is available of Dr. Jon Beever's presentation given during the Angus Education Center, held in conjunction with the Association's 125th Annual Meeting in Louisville, KY. To view the video, click here.

For a complete listing of Arthrogryposis Multiplex (AM) information released by the American Angus Association click here.

Fawn Calf Syndrome

David Steffen DVM Ph.D., University of Nebraska

As we continue our work to characterize and head off emerging problems, we are also carefully monitoring a non-lethal condition referred to as fawn calf syndrome (FCS). Calves suspected to have FCS have been reported in Angus calves in Australia for many years and researchers there have identified the condition as an inherited trait allegedly tracing to U.S. sires. If the Australian hypothesis is correct, and there is strong evidence that it is, there are almost certainly cases unintentionally not being reported in the United States. FCS calves are normally born alive and most can walk, suckle and survive. The birth weight of FCS calves is normal.

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Photos of calves afflicted with Fawn Calf Syndrome

Video: Fawn calf syndrome in angus calves

Note: There is no audio track in this video
Footage of the symptoms of fawn calf syndrome in angus calves as well as information on clinical features, genetics and clinical progression at the end of the video

Hydrocephalus


By David Steffen DVM Ph.D., University of Nebraska
Since arthrogryposis multiplex (AM) was first reported by the American Angus Association as a possible inherited defect, breeders have stepped forward with great cooperation to report abnormal calves. This allowed Dr. Beever and I to characterize the phenotype, identify the gene and to develop a test. This success was contingent on your cooperation for which we are thankful.

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